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    §ImmuneResearch protocol

    Thymosin Alpha-1.

    Thymosin Alpha-1 (Tα1) is a 28–amino acid peptide originally isolated from the thymus gland, recognized for its broad immunomodulatory properties[1]. It has been investigated as an immune enhancer ...

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    Vial Size:
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    Calculated-volume support unavailable

    This selected cited guide does not contain one unambiguous vial, per-event amount, cadence, and diluent set. No value was inferred from general library metadata. Review the cited table below before creating a private Research Use Only record.

    Cited protocol & reconstitution guide

    Source-backed reference fields by phase, including any volume fields authored in the cited guide.

    Standard adult (≥40 kg)

    1.6 mg per injection

    Units / volume32 units (0.32 mL)

    Adult under 40 kg — 30 kg body weight

    1.2 mg

    Units / volume24 units (0.24 mL)

    Adult under 40 kg — 35 kg body weight

    1.4 mg

    Units / volume28 units (0.28 mL)

    Reconstitution by vial size

    Calculated volume for each cited phase and vial variant. The highlighted column matches the selection above.

    Phase
    5 mg
    1 mL water
    10 mg
    2 mL water
    Standard adult (≥40 kg)
    32 u
    0.32 mL
    32 u
    0.32 mL
    Adult under 40 kg — 30 kg body weight
    24 u
    0.24 mL
    24 u
    0.24 mL
    Adult under 40 kg — 35 kg body weight
    28 u
    0.28 mL
    28 u
    0.28 mL

    Overview

    Overview

    Thymosin Alpha-1 (Tα1) is a 28–amino acid peptide originally isolated from the thymus gland, recognized for its broad immunomodulatory properties[1]. It has been investigated as an immune enhancer in chronic viral infections (hepatitis B/C, HIV/AIDS) and critical illness (sepsis, COVID-19)[2][3]. This educational protocol presents a once‑daily subcutaneous approach using a practical dilution for clear insulin‑syringe measurements. Reconstitute: Add 3.0 mL bacteriostatic water → ~1.67 mg/mL conc

    Category
    Immune
    Routes
    subcutaneous

    Mechanism

    Thymosin Alpha-1

    Mechanism of action

    Mechanism of action

    TA1 is a signaling peptide. It does not kill viruses or cancer cells directly. Instead, it tunes the immune system so the body's existing defenses work more effectively. The pathway is well-mapped. TA1 binds two pattern-recognition receptors on antigen-presenting cells - Toll-like receptor 2 (TLR2) and Toll-like receptor 9 (TLR9) - which are the same receptors the immune system uses to detect bacterial and viral DNA. Activating them mimics the signal of a real pathogen and primes the rest of the immune response. Downstream of TLR2 and TLR9, three things happen: dendritic cells mature into effective antigen presenters, T cells differentiate into helper (CD4+) and cytotoxic (CD8+) populations, and the immune balance shifts toward Th1 , which is the side of immunity that handles intracellular infections (like hepatitis viruses) and tumor surveillance. Importantly, TA1 is described in the literature as a modulator rather than a pure stimulator. Across decades of clinical use it does not appear to produce the cytokine-storm-style overactivation seen with some other immune therapies, which is one reason its safety profile has held up across so many indications.

    Key research findings
    • 01

      Identity / origin (animal-model foundation): Thymosin alpha-1 (Tα1; also called thymalfasin) is a 28-amino-acid peptide originally isolated from thymic tissue (thymosin fraction 5) and identified as the factor that restored immune function in thymectomized animals (reviewed by King & Tuthill, 2016).

    • 02

      Mechanism (in vitro / cell-based): Characterized as a pleiotropic immune-response modifier that signals through Toll-like receptors (e.g., TLR2/TLR9) on myeloid and plasmacytoid dendritic cells, influencing T-cell maturation and Th1-type cytokine production.

    • 03

      Animal models: Studied for effects on immune-cell subsets, dendritic-cell activation, responses to infection, and as a vaccine adjuvant; reported activity is framed as immunomodulatory rather than directly antimicrobial.

    • 04

      Human studies: Evaluated in clinical trials in immune-suppression and chronic viral-infection contexts and marketed as thymalfasin in several countries; the consistency and quality of controlled human efficacy data vary by context.

    • 05

      Human (observational, COVID-19): In a retrospective, non-randomized analysis of hospitalized severe COVID-19 patients (Liu et al., 2020), Tα1 administration was associated with changes in circulating CD4+/CD8+ T-cell counts and T-cell exhaustion markers (PD-1, Tim-3); interpretation is limited by the observational design.

    Primary source: Chronic hepatitis B (strongest evidence): Pooled analysis of three RCTs in the Zadaxin label found sustained virologic response advantages over placebo with 1.6 mg twice weekly for 6 months, especially when measured 12 months after the end of therapy. This is the foundation of TA1's international approval. Chronic hepatitis C with interferon (moderate): Pooled analysis of 2 RCTs and 1 historical-controlled trial showed sustained ALT normalization in 22.4% of TA1 + interferon patients versus 9.3% with interferon alone. Newer DAA-based hepatitis C regimens have largely replaced interferon, so the practical relevance has narrowed. Cancer chemotherapy adjunct (moderate, fragmented): Smaller RCTs and observational studies report reduced infection rates and better immune-cell preservation during chemotherapy in hepatocellular carcinoma, NSCLC, and melanoma cohorts. Trials are inconsistent in design, and a single landmark RCT does not yet exist. Severe sepsis (mixed - read carefully): The 2025 BMJ TESTS trial (1,106 adults, 22 Chinese centers, double-blinded, placebo-controlled) was the largest TA1 sepsis study ever conducted. The primary endpoint - 28-day all-cause mortality - was not significantly different (hazard ratio 0.94, 95% CI 0.76-1.16, p=0.54). A 2025 Frontiers meta-analysis of 11 RCTs (n=1,927) reported a pooled odds ratio of 0.73 for 28-day mortality, but the authors flagged heterogeneity and credibility caveats. Many secondary write-ups oversimplify this. Post-COVID and long COVID (early): A 2023 meta-analysis of COVID-19 trials found mortality reduction in some patient subgroups but no consistent length-of-stay improvement. Dedicated long COVID trials are limited. General immune support / anti-aging (mechanism, not trials): Thymic involution with age is real, and TA1 is mechanistically a thymic peptide, but dedicated trials in healthy adults using TA1 for anti-aging endpoints are limited. Most community use rests on the chronic-disease trial base. Read the BMJ 2025 sepsis paper carefully

    Pharmacokinetic profile

    Literature reference (RUO)

    Single-dose plasma curve over 24h. Shaded band = commonly-cited therapeutic window. Illustrative only.

    Researched Effects

    Researched benefits

    Areas of active research and investigation. Results may vary and are based on preclinical or early clinical data.

    ✨

    Supports enhanced T‑cell function and overall immune competence[1][2].

    ✨

    Demonstrates an excellent safety profile; doses up to 1.6 mg twice weekly for 6–12 months have been well-tolerated[4][7].

    ✨

    Even at experimental doses up to 16 mg SC over 12 months, no significant Tα1‑specific toxicity has been observed[7].

    Protocol Reference

    Protocol reference

    Research Use Only. PeptiJournal supports private research documentation and calculation support. It does not provide medical advice, human-use directions, or claims of safety or effectiveness.
    subcutaneous

    Commonly cited research range: 300–500 mcg, daily.

    Reference figures reported in the research literature — not a dosing recommendation. For interactive vial math and scheduling, see the Calculator and Schedule tabs.

    Cited protocol & reconstitution guide

    Source-backed reference fields by phase, including any volume fields authored in the cited guide.

    Standard adult (≥40 kg)

    1.6 mg per injection

    Units / volume32 units (0.32 mL)

    Adult under 40 kg — 30 kg body weight

    1.2 mg

    Units / volume24 units (0.24 mL)

    Adult under 40 kg — 35 kg body weight

    1.4 mg

    Units / volume28 units (0.28 mL)

    Titration protocol

    1. Standard adultStart
      1.6 mg per injection

      Subcutaneous. Twice weekly, injections spaced 3-4 days apart (e.g., Monday and Thursday). Rotate sites — abdomen, thigh, or upper arm. A twice-weekly dose missed by 1-2 days is typically resumed on schedule without doubling up.

    2. Adult under 40 kg — weight-adjustedBuild
      40 mcg/kg

      Body-weight-adjusted rule for adults weighing under 40 kg (88 lb). Same twice-weekly subcutaneous schedule. At 5 mg/mL this works out to 0.008 mL (0.8 units) per kg of body weight.

    3. Adult under 40 kg — 30 kg body weightBuild
      1.2 mg

      Twice weekly. Labeled an approximate dose in the source table.

    4. Adult under 40 kg — 35 kg body weightBuild
      1.4 mg

      Twice weekly. Labeled an approximate dose in the source table.

    5. Adult ≥40 kg body weightBuild
      1.6 mg

      Standard adult. Twice weekly.

    6. Cycle reference — 6 monthsBuild
      1.6 mg

      Twice weekly across the full run.

    7. Cycle reference — 6-12 monthsBuild
      1.6 mg

      Twice weekly across an extended-duration run.

    8. Cycle reference — 7 days continuousBuild
      Not stated as a mg value

      Every 12 hours for 7 days. The source table gives the schedule only, with no mg value.

    9. Cycle reference — 4-12 weeksMaintenance
      Not stated as a mg value

      Community-derived pattern, not drawn from a controlled trial. A separately noted community pattern is 1 mg daily for 10-30 days, also not from controlled trials.

    Storage & Handling

    Storage requirements(typical for most peptides)

    ❄️
    Lyophilized (powder)
    -20°C (frozen)

    Can be stored for extended periods. Protect from moisture.

    🧊
    Reconstituted
    2-8°C (refrigerated)

    Store in refrigerator door. Never freeze after reconstitution.

    ⏱️
    Stability window
    28-30 days after reconstitution

    Label vials with reconstitution date. Discard if cloudy.

    Reconstitution steps

    1. 01🌡️01 Inspect the vial — Confirm the label, vial size, lot number, and that the powder pellet looks intact and dry.
    2. 02🧴02 Choose your concentration — Match a row from the reconstitution table above to the vial size you have and the syringe size you plan to use.
    3. 03💉03 Prep the surfaces — Wipe the rubber stopper of both the BAC water vial and the TA1 vial with a fresh alcohol swab. Let them dry.
    4. 04💧Draw 1 mL bacteriostatic water into a sterile syringe — this 5 mg vial yields 5 mg/mL.
    5. 05🔄05 Inject the BAC water — Insert the needle into the TA1 vial and push the BAC water down the inside wall of the vial, not directly onto the powder pellet.
    6. 06🏷️06 Mix gently — Swirl the vial slowly until the powder dissolves. Do not shake. The solution should be clear with no visible particles.
    7. 07❄️07 Label and refrigerate — Write the reconstitution date on the vial and store at 2-8°C (35.6-46.4°F). Use within 30 days.
    8. 08💉Important: This guide is for educational purposes only and is not medical advice. For research use only. Not for human consumption.

    Additional storage notes

    Lyophilized (powder form)

    35.6-46.4°F (2-8°C) long-term — Short-term room temperature is acceptable per the Zadaxin label, but refrigeration extends shelf life. Use the manufacturer's expiration date as the long-term reference.

    Reconstituted (liquid form)

    35.6-46.4°F (2-8°C) — Use within 30 days. Avoid freezing - freeze-thaw cycles degrade the peptide.

    Light exposure

    Protect from direct light — Store in original packaging or a closed compartment.

    Travel

    Insulated cooler with cold pack — Avoid letting the vial sit at room temperature for extended travel periods. Do not freeze.

    Clinical Evidence

    Clinical evidence

    Investigated in immunology research and clinical studies for immune modulation in infection and vaccine-adjuvant contexts.

    Chronic hepatitis B (strongest evidence): Pooled analysis of three RCTs in the Zadaxin label found sustained virologic response advantages over placebo with 1.6 mg twice weekly for 6 months, especially when measured 12 months after the end of therapy. This is the foundation of TA1's international approval. Chronic hepatitis C with interferon (moderate): Pooled analysis of 2 RCTs and 1 historical-controlled trial showed sustained ALT normalization in 22.4% of TA1 + interferon patients versus 9.3% with interferon alone. Newer DAA-based hepatitis C regimens have largely replaced interferon, so the practical relevance has narrowed. Cancer chemotherapy adjunct (moderate, fragmented): Smaller RCTs and observational studies report reduced infection rates and better immune-cell preservation during chemotherapy in hepatocellular carcinoma, NSCLC, and melanoma cohorts. Trials are inconsistent in design, and a single landmark RCT does not yet exist. Severe sepsis (mixed - read carefully): The 2025 BMJ TESTS trial (1,106 adults, 22 Chinese centers, double-blinded, placebo-controlled) was the largest TA1 sepsis study ever conducted. The primary endpoint - 28-day all-cause mortality - was not significantly different (hazard ratio 0.94, 95% CI 0.76-1.16, p=0.54). A 2025 Frontiers meta-analysis of 11 RCTs (n=1,927) reported a pooled odds ratio of 0.73 for 28-day mortality, but the authors flagged heterogeneity and credibility caveats. Many secondary write-ups oversimplify this. Post-COVID and long COVID (early): A 2023 meta-analysis of COVID-19 trials found mortality reduction in some patient subgroups but no consistent length-of-stay improvement. Dedicated long COVID trials are limited. General immune support / anti-aging (mechanism, not trials): Thymic involution with age is real, and TA1 is mechanistically a thymic peptide, but dedicated trials in healthy adults using TA1 for anti-aging endpoints are limited. Most community use rests on the chronic-disease trial base. Read the BMJ 2025 sepsis paper carefully

    1. 01Identity / origin (animal-model foundation): Thymosin alpha-1 (Tα1; also called thymalfasin) is a 28-amino-acid peptide originally isolated from thymic tissue (thymosin fraction 5) and identified as the factor that restored immune function in thymectomized animals (reviewed by King & Tuthill, 2016).
    2. 02Mechanism (in vitro / cell-based): Characterized as a pleiotropic immune-response modifier that signals through Toll-like receptors (e.g., TLR2/TLR9) on myeloid and plasmacytoid dendritic cells, influencing T-cell maturation and Th1-type cytokine production.
    3. 03Animal models: Studied for effects on immune-cell subsets, dendritic-cell activation, responses to infection, and as a vaccine adjuvant; reported activity is framed as immunomodulatory rather than directly antimicrobial.
    4. 04Human studies: Evaluated in clinical trials in immune-suppression and chronic viral-infection contexts and marketed as thymalfasin in several countries; the consistency and quality of controlled human efficacy data vary by context.
    5. 05Human (observational, COVID-19): In a retrospective, non-randomized analysis of hospitalized severe COVID-19 patients (Liu et al., 2020), Tα1 administration was associated with changes in circulating CD4+/CD8+ T-cell counts and T-cell exhaustion markers (PD-1, Tim-3); interpretation is limited by the observational design.

    Evidence maturity varies by compound; much peptide research is preclinical (in vitro or animal-model). Where human data are limited, findings should be read as research observations, not clinical conclusions.

    References

    Literature references

    Published research articles and sources related to Thymosin Alpha-1.

    1. 01
      Wu J, Pei F, Zhou L, et al. The efficacy and safety of thymosin α1 for sepsis (TESTS): multicentre, double blinded, randomised, placebo controlled, phase 3 trial. BMJ (2025)
      et al. (2025)
    2. 02
      Gu B, Zhou Y, Nie Y, et al. Efficacy of thymosin α1 for sepsis: a systematic review and meta-analysis of randomized controlled trials. Frontiers in Cellular and Infection Microbiology (2025)
      et al. (2025)
    3. 03
      Dominari A, et al. Thymosin alpha 1: A comprehensive review of the literature. World Journal of Virology (2020)
      et al. (2020)
    4. 04
      Tao N, et al. Thymosin α1 and Its Role in Viral Infectious Diseases: The Mechanism and Clinical Application. Molecules (MDPI) (2023)
      et al. (2023)
    5. 05
      Dinetz E, Lee E. Comprehensive Review of the Safety and Efficacy of Thymosin Alpha 1 in Human Clinical Trials. Alternative Therapies in Health and Medicine (2024)
      et al. (2024)
    6. 06
      SciClone Pharmaceuticals Zadaxin (thymalfasin) Professional Monograph - dosing, indications, and pregnancy category. SciClone / peptidesociety.org archive (2018)
      et al. (2018)
    7. 07
      Rost K, Wierich W, Masayuki F, Tuthill C, Horwitz D, Herrmann W. Pharmacokinetics of thymosin alpha 1 after subcutaneous injection of three different formulations in healthy volunteers. International Journal of Clinical Pharmacology and Therapeutics (1999)
      et al. (1999)
    8. 08
      U.S. Food and Drug Administration Bulk Drug Substances Nominated for Use in Compounding Under Section 503A - 503A bulks list updates and Category 2 status. FDA (2024)
      et al. (2024)
    9. 09
      U.S. Food and Drug Administration PCAC Topic 1: thymosin alpha-1-related bulk drug substances - December 4, 2024 transcript. FDA Pharmacy Compounding Advisory Committee (2024)
      et al. (2024)
    10. 10
      Lexology / Hyman Phelps & McNamara FDA removes certain peptide bulk drug substances from Category 2 of interim 503A bulks list and sets dates for PCAC review. Lexology (2024)
      et al. (2024)
    11. 11
      Tian Y, et al. Thymosin alpha 1 alleviates inflammation and prevents infection in patients with severe acute pancreatitis through immune regulation: a systematic review and meta-analysis. Frontiers in Immunology (2025)
      et al. (2025)
    12. 12
      Zadaxin Drug Description / RxList compilation ZADAXIN thymosin alpha 1 (thymalfasin) - indications, dosing, and ALT flare label note. RxList / SciClone label compilation (2024)
      et al. (2024)
    Search PubMed for Thymosin Alpha-1

    Observed Effects

    Observed effects in cited research

    Reported observations
    • Injection-site reaction — Common — Mild redness, soreness, or swelling that resolves within hours. Rotate sites.
    • Transient ALT flare (liver enzyme bump) — Common in HBV/HCV use — An ALT rise to >2x baseline can occur during therapy; per the Zadaxin label, treatment is generally continued unless signs of liver failure appear.
    • Mild transient fatigue — Occasional — Usually first few injections as immune activation begins.
    • Mild flu-like symptoms — Occasional — Reflects immune-system activation, not infection.
    • Headache — Rare — Mild and transient; resolves with hydration.
    • GI discomfort — Rare — Mild stomach upset reported in a minority of users.
    • Polyarthralgia with hand edema — Very rare — Joint pain with hand swelling reported in isolated cases.
    • Hypersensitivity / allergic reaction — Very rare — Limited to individuals with known peptide sensitivities.
    Drug-interaction profile
    • TA1 has a notably clean drug-interaction profile. The Zadaxin label reports no clinically significant interactions in published trials and notes compatibility with antiretroviral therapy, conventional hepatitis treatments, and most antimicrobial regimens. Never mix TA1 with other drugs in the same syringe - always inject separately.
    • The one place to slow down is when TA1 is being considered alongside other immune-modulating drugs (immunosuppressants, biologics, or other immune enhancers). Additive effects are theoretically possible and warrant clinician oversight.
    Quality-control risk
    • TA1 is a research-use product in the US. Vial-to-vial purity and content can vary across suppliers. A current Certificate of Analysis (COA) and a clear product label are the minimum quality signals to look for.

    Research Considerations

    Research considerations

    Research Use Only - not for human or veterinary therapeutic use. Has been studied in clinical research; on this platform it is handled strictly as a research material. Consult a licensed healthcare professional for any clinical decisions.

    • TA1 has a benign safety record across more than three decades of international clinical use, but the same immune-activating mechanism that makes it useful also creates clear groups of people who should not use it without specialist oversight.
    • Organ transplant recipients on immunosuppressive therapy. TA1 enhances cell-mediated immunity, which is the exact arm of immunity that anti-rejection drugs are trying to suppress. Combining the two could undermine transplant medication or trigger rejection.
    • Adults with active autoimmune flares. Because TA1 amplifies T-cell activity, it could in theory worsen an active autoimmune attack. Some clinical reports suggest stable, well-controlled autoimmune patients tolerate TA1, but starting the peptide during a flare is not supported by the evidence base.
    • Pregnant or breastfeeding women. Human safety data in pregnancy and lactation is limited. Animal studies have not shown fetal harm, but the Zadaxin label categorizes the drug as Category C and recommends use only when benefits clearly outweigh risks.
    • Adults under 18. Pediatric safety has not been established in published trials.
    • Anyone with known hypersensitivity to TA1, thymalfasin, or any vial component.

    Factors noted in the research literature; not patient-specific medical advice.

    Regulatory Status

    Regulatory status

    RUO

    Research Use Only. PeptiJournal supports private research documentation and calculation support. It does not provide medical advice, human-use directions, or claims of safety or effectiveness.

    Comparisons

    Comparisons

    CompoundMechanismRouteStatus
    Thymosin Alpha-1thisA 28-amino-acid thymic peptide that modulates immune function, studied for enhancement of T-cell maturation, dendritic-cell function, and Toll-like-receptor signaling.subcutaneousInvestigational / RUO
    TirzepatideActivates both GIP and GLP-1 receptors, providing synergistic effects on insulin secretion, glucose control, and appetite regulation.subcutaneousInvestigational / RUO
    Vesugen (Lys-Glu-Asp)A synthetic tripeptide bioregulator (Lys-Glu-Asp) studied for gene-regulatory support of vascular endothelial tissue.subcutaneousInvestigational / RUO
    VilonA synthetic immunoregulatory dipeptide (Lys-Glu) studied for gene-regulatory and immunomodulatory activity, including effects on lymphocyte markers and chromatin structure.subcutaneousInvestigational / RUO
    5-Amino-1MQInhibits nicotinamide N-methyltransferase (NNMT), increasing NAD+ availability and enhancing mitochondrial metabolism.oral, subcutaneousInvestigational / RUO
    AdipotideA peptidomimetic that targets prohibitin on the vasculature of white adipose tissue and delivers a pro-apoptotic sequence, studied for selective reduction of fat-tissue blood supply.subcutaneousInvestigational / RUO
    AICARA cell-permeable nucleoside phosphorylated intracellularly to ZMP, an AMP-mimetic that activates AMP-activated protein kinase (AMPK), shifting cells toward oxidative metabolism.subcutaneousInvestigational / RUO

    Attributes shown for research comparison only; not a statement of efficacy or therapeutic equivalence.

    FAQ

    Frequently asked questions

    Research-use notice

    Research Use Only. This educational content and calculation support is intended for private research documentation. It does not provide medical advice, human-use directions, or claims of safety or effectiveness.

    Cited guide source: View source

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